Education
Ph.D., North Carolina, 1973
B.A., SUNY at Binghamton, 1967

Recent Publications

Crowley, J.J, Treistman, S.N., and Dopico, A.M. (2005) Distinct structural features of phospholipids differentially determine ethanol sensitivity and basal function of BK channels, (In press, Mol. Pharmacol.).

Newton PM, Tully K, McMahon T, Connolly J, Dadgar J, Treistman SN, Messing RO. (2005) Chronic ethanol exposure induces an N-type calcium channel splice variant with altered channel kinetics. FEBS Lett. Jan 31;579(3):671-6.

Yuan C, O’Connell R, Feinberg-Zadek, P, Johnston L, and Treistman SN (2004)  Bilayer thickness modulates the conductance of the BK channel in model membranes. (Biophysical Journal, 86(6):3620-33).

Tully, K. and Treistman, (2004) S.N. Distinct Intracellular Calcium Profiles Following Influx Through N vs L Type Calcium Channels: Role of Ca2+-induced Ca2+ release. J. Neurophysiol. 92(1):135-43.

Martin, G., Puig, S.I., Pietrzykowski, A., Zadek, P.,Emery, P., and Treistman. S.N. (2004) Restricted cellular localization of a specific BK-channel subtype controls ethanol sensitivity in the nucleus accumbens, J. Neurosci. 24(29):6563-72).

Pietrzykowski, A., Martin, G., Puig, S. and Treistman, (2004) S.N. Alcohol Tolerance in BK Channels of Neuronal Terminals is Intrinsic, and Includes Two Components: Decreased Channel Sensitivity to Ethanol and Decreased Channel Density (J. Neuroscience, J Neurosci. 24(38):8322-32. 

Levine JB, Martin G, Wilson A, Treistman SN (2003)  Clozapine inhibits isolated N-methyl-D-aspartate receptors in a subunit specific manner. Neurosci. Lett. 346:125-128

Crowley JJ, Treistman SN, and Dopico AM (2003) Cholesterol antagonizes ethanol potentiation of human brain BKCa channels reconstituted into phospholipid bilayers Mol. Pharmacol. 64:365-372.

Ortiz-Miranda, S., Dayanithi, G., Coccia, V., Custer, E., Alphandery, E., Mazuc, E., Treistman, S.N., Lemos, J.R. (2003) Mu-opiod receptor modulates peptide release from rat neurohypophysial terminals by inhibiting Ca2+ influx. J. Neuroendocrinol. 15:888-894. 

Treistman, S.N., O’Connell, R.M., and Crowley, J. (2002) Artificial bilayer techniques for study of alcohol action on ion channels. In: Methods for Alcohol-Related Neuroscience Research; CRC Press.

Knott, T., Dayanithi, G., Lemos, J.R., Dopico, A.M., Treistman, S.N. (2002) Integrated channel plasticity contributes to alcohol tolerance in neurohypophysial terminals. Mol. Pharmacol. 62:135-142.

Knott, T., Dayanithi, G., Custer, E., Lemos, J.R., Dopico, A.M., Treistman, S.N. (2000) Tolerance to acute ethanol inhibition of hormone release in the isolated neurohypophysis, Alc:Clin. Exp. Res. 24:1077-1083.

Tully, K.,  Kupfer, D., Dopico, A.M., S.N. Treistman (2000) A plasticizer  released from i.v. drip chambers elevates calcium levels in neurosecretory terminals. Toxicol.Appl. Pharmacol. 168:183-188.

Treistman, S.N. (2000) Addiction. Encyclopedia of Life Sciences. Macmillan Press.

Treistman, S.N., Mullikin- Kilpatrick, D., Roche, J. (2000) G protein mediation of ethanol effects on calcium channels. In: Ethanol and intracellular signaling: From molecules to behavior.

Crowley, J.J., Dopico, A.M., and Treistman, S.N. Role of phosphatidylserine in making BK channels compliant for ethanol potentiation does not involve charge (Submitted).

Dopico, A., Widmer, H., Lemos, J.R., Treistman, S.N. (1999) Rat supraoptic neurones show distinct large conductance, Ca2+ -activated K channel subtypes in cell bodies versus nerve endings. J. Physiol. 519:101-114.

Dopico, A.M. Chu, B., Lemos, J.R., Treistman, S.N. (1999) Alcohol modulation of calcium-activated potassium channels. Neurochemistry International 35:103-106.

Dopico, A., Widmer, H., Lemos, J.R., Treistman, S.N. (1999) Rat supraoptic neurones show distinct large conductance, Ca2+ -activated K+ channel subtypes in cell bodies versus nerve endings. J. Physiol. 519:101-114.

Other Selected Publications

Dopico, A., Widmer, H., Lemos, J.R., Treistman, S.N. (1999) Rat supraoptic neurones show distinct large conductance, Ca2+ -activated K+ channel subtypes in cell bodies versus nerve endings. J. Physiol. 519: 101-114.

Treistman, S.N., Chu, B., Dopico, A. (1999) Molecular targets underlying ethanol-mediated reduction of hormone release from neurohypophysial nerve terminals. In: Y. Liu and W. Hunt eds., Plenum Press, The Drunken Synapse , Ch. 3, pp. 27-38.

Dopico, A.M. Chu, B., Lemos, J.R., Treistman, S.N. (1999) Alcohol modulation of calcium-activated potassium channels. Neurochemistry International., 35: 103-106.

Roche, J. and Treistman, S.N. (1998) The Ca2+ channel b3 subunit differentially modulates the sensitivity of a1A and a1B Ca2+ channels to muscarinic M2 mediated G protein inhibition. J. Neurosci. 18: 878-886.

Dopico, A., Anantharam, V., and Treistman, S.N. (1998) Ethanol increases the activity of Ca++ -dependent K+ (mslo) channels: Functional interaction with cytosolic Ca++. J. Pharm. Exp. Ther. 284: 258-268.

Roche, J. and Treistman, S.N. (1998) Ca2+ channel b3 subunit enhances voltage-dependent relief of G protein inhibition induced by muscarinic receptor activation and Gbg J. Neurosci. 18: 4883-4890.

Widmer, H., Lemos, J.R., and Treistman, S.N. (1998) Ethanol reduces the duration of single evoked spikes by a selective inhibition of voltage-gated calcium currents in acutely-dissociated supraoptic neurons of the rat. J. Neuroendocrin. 10: 399-406.

Chu, B., Dopico, A.M., Lemos, J.R., and Treistman, S.N. (1998) Ethanol potentiation of calcium-activated potassium channels reconstituted into planar lipid bilayers. Mol. Pharmacol. 54: 397-406.